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Here, we review the historical progress in the development of EGFR TKIs and on-target-mediated and EGFR -independent resistance mechanisms, aiming to summarize the current status of and provide future directions for EGFR TKI research

Decreased activity of Cu-dependent enzymes, such as SOD, and decreased levels of ceruloplasmin in patients with ME/CFS [371] can be explained by a deficiency in Cu uptake due to overexpression of MTs in enterocytes in response to mucosal infection

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