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Notably, L2HG-induced ferroptosis occurs independently of impaired cystine uptake, transsulfuration pathway activity, or increased GPX4-mediated GSH utilization, revealing a non-canonical ferroptosis mechanism driven by enhanced GSH catabolism
These interfacial residues in Ube2e2 were identified as highly conserved among Ube2e family members, but were absent in Ube2d3 (Fig
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