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There is much interest in its clinical application in various neural disorders such as Alzheimer's disease and painful neuropathies [114]
Ir J Med Sci 193 , 16211639 (2024)
When tissue folate and cobalamin stores are intact, an abnormal increment of postmethionine-load homocysteinemia may also reveal a saturation of the transsulfuration pathway caused by dietary methionine excess that might be combined with transsulfuration defects (in cases with basal hyperhomocysteinemia) or with the homozygous thermolabile MTHFR defect (in cases with normal fasting homocysteine) (Cattaneo, 1999)
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