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Taken together, these findings suggest that glutathione synthesis within TLS may be distinctly regulated, with dendritic cells and fibroblasts that express the cystine/glutamate transporter playing an important role
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Further isotope labeling experiments confirmed that upon GGCT overexpression, Gln is preferentially shunted to the GSH synthesis pathway, with a significantly reduced proportion entering the TCA cycle
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It triggers growth hormone release with typically mild impact on cortisol and prolactin levels which helps avoid stress or unwanted effects
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