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Therefore, in the present study, we examined the relative contributions of eNOS to gastric ulcer healing after BPC 157 treatment in rats and, as expected, the AKT phosphorylation, p38/MAPK dephosphorylation and the phosphorylated eNOS expression was increased significantly compared to Clopidogrel group

Multifaceted roles of HSF1 in cell death: a state-of-the-art review

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