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Conversely, reversal of GLP-1(936)s glucagonostatic effect might explain the dramatic elevation of circulating glucagon observed after pharmacological/genetic inhibition of the GCGRs [43, 53, 65, 66], an effect that has been attributed to AA-induced stimulation of alpha cell proliferation
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DESeq2 was then applied on the pseudo-bulk gene expression matrix with treatment group and time point post-PBS or LPS treatment as variables in the design matrix
We then isolated the -cells from each group for further analysis (S5B-S5C)
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