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Normally microglia are neuroprotective, cleaning out debris that may contribute to neurodegenerative diseases or damage from ischemic stroke, but in the case of TBI they can become overactivated and exacerbate neuroinflammation (with upregulated TNFa, IL-6, C3, NK-kB, CD68, etc), and vascular changes can result in headaches, neurovascular flow changes, impaired lymphatic flow, blood-brain barrier disruption, activation of pro-inflammatory pathways, neuronal death, hyperexcitability, and disrupted synaptic connections

Therapeutic strategies targeting mitochondrial OS, known as mitochondrial-targeted antioxidants (MTAs) such as MitoQ, are modulators and promoters of mitochondrial biogenesis (PGC-1 activators), which have shown promise in preclinical HTN models (Halling et al., 2019)

Phytomedicine 84, 153519

K., Wang, Y

Two procedures are performed during distinct patient encounters on the same day