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Mechanism appears multifactorial: upregulation of nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), and synaptic plasticity proteins like synaptophysin
The disulfide bridge creates a ring structure at the N-terminus that positions the adjacent residues for productive engagement with the extracellular binding domain of the amylin and calcitonin receptors
Neuroprotective effect of caffeine in Alzheimers disease
Future research should focus on large-scale, well-controlled randomized trials with standardized dosing protocols, clearly defined deficiency thresholds, and long-term follow-up to strengthen the evidence base and guide clinical practice
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