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Iron binding proteins such as transferrin and ferritin may contribute to keep free Fe (III) concentration in the biological fluid low [44]

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Such accumulation would sequester more amino acids, would increase transport out of the cell up to saturation, and would therefore increase the loss of cysteine to cystine in the blood

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This suggests BPC-157 modulates the angiogenic response through pathway interactions rather than acting as a direct angiogenic factor