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The critical involvement of lysosomal damage in different pathways of cell death has been widely studied, but the current focus is placed on the release of cathepsin-based contents caused by structural damage

[1] This discomfort is usually transient and can be minimized by using proper technique and allowing the solution to come to room temperature before injection

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Encouragingly, many EDS patients who have carefully weighed these factors and proceeded with therapy have found the benefits to outweigh the downsides, reporting that improved stability and pain control gave them back a measure of function and quality of life that they couldnt achieve otherwise