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d , Nedd8 K0 (no lysine residue) mutant inhibited PDK1 neddylation in cells

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This trend, however, was not found to correlate with the turn-over rate of the mutant (Table 2)

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At the same time, concurrent GLP-1 and GIP activity may help offset the glucose-raising effects associated with glucagon alone.[1][3] Ongoing Phase 3 research will help clarify how this combined mechanism translates into clinical outcomes