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While the inflammation-reducing and fibrosis-attenuating effects of glucagon agonism are less well-characterized, the reductions in hepatic inflammation parameters and fibrosis observed in preclinical models likely reflect secondary effects consequent to improved steatosis, rather than direct anti-inflammatory actions [21]

"And I'm kind of like, 'Well, it's what I'm doing now

Tuttle, Mechanisms and clinical applications of incretin therapies for diabetes and chronic kidney disease

That's a more specific question - and it has a more specific answer than most comparison guides give you

Das liegt zu einem Groteil an der ungesunden Ernhrungs- und Lebensweise der Menschen