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Like other G protein-coupled receptors, the GLP-1R undergoes agonist-mediated endocytosis, but the functional and therapeutic consequences of modulating GLP-1R endocytic trafficking have not been clearly defined
This dual-agonism approachtargeting both GIP and GLP-1seems to work better than the single-pathway method used by semaglutide
BPC 157 enhanced the phosphorylation of Src, Cav-1 and eNOS which was abolished by pretreatment with Src inhibitor, confirming the upstream role of Src in this signal pathway
Calcif Tissue Int (2015) 97(5):50615
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