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Using nanoBRET technology, we focused on ligand-stimulated receptor recruitment of G i and -arrestin2, initiators of G protein-dependent and G protein-independent signaling pathways, respectively 56
When administered together, their mechanisms converge to amplify appetite regulation, energy intake reduction, and glycemic improvements, exceeding the individual effects of monotherapy by acting on complementary neuroendocrine circuits regulating body weight and metabolism 1-3
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