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Rather than promoting growth directly, it aims to clear dysfunctional cellular components that impede normal tissue renewal processes
Published research has documented that low NAD+ levels slow DNA repair by up to 40% (through impaired PARP function), reduce sirtuin-mediated gene regulation (through substrate depletion), compromise mitochondrial electron transport efficiency (through reduced NADH availability), and promote cellular senescence (through the combined effects of genomic instability, metabolic dysfunction, and impaired stress responses)
Some researchers use higher doses up to 1000mcg per administration when targeting more aggressive recovery goals
