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Further human studies are required to substantiate the claims
9 Conclusion and future prospects The pathogenesis of ALD is inextricably linked to dysregulation of OS-related signaling networks, involving dynamic interactions among key molecules such as SREBP, Nrf2, and AMPK
Clinical description All patients with gamma-glutamylcysteine synthetase deficiency have had hemolytic anemia, usually rather mild [18]
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