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High expression of HNF4 in EC may supplement intracellular GSH through a non-xCT-dependent pathway by activating metabolic enzymes involved in methionine metabolism, which may represent a key mechanism underlying ferroptosis resistance in EC
Nature Immunology , 25 (3), 483495
-GluGln belongs to the -glutamyl dipeptide, which is usually derived from extracellular GSH degradation
J.YangW
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