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Under glucose deprivation or increased ATP demand, AMPK activation is expected to shift oligodendrocytes from an anabolic, myelin-maintenance program toward a catabolic state that supports survival
This results in inflammation of both tissue and joints
In addition, the analysis of human prostate cancer and prostate control specimens confirmed the aberrant expression of miR-124-3p, miR-129-5p, and miR-378 in primary tumors
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M2 macrophage hybrid membrane-camouflaged targeted biomimetic nanosomes to reprogram inflammatory microenvironment for enhanced enzyme-thermo-immunotherapy
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