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This glucose-dependent mechanism is fundamentally different from older diabetes medications like sulfonylureas, which stimulate insulin regardless of blood sugar levels and carry a significant risk of hypoglycemia
We considered that GSTM2 was likely be more stable than GSTM1 from a genetic perspective, and therefore, up-regulating GSTM2 may be a better choice for inducing protective effects
The OCR even mentioned studies showing it reduced body weight, BMI, and fat mass which aligns with what I was hoping for
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Bai R, Guo J, Ye X, Xie Y, Xie T
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