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As previously discussed, neurodegenerative and autoimmune disorders such as AD, MS, and PD are associated with reduced GSH concentrations in the brain making supplemental GSH an attractive therapy if the limitations in delivery can be overcome (Smeyne and Smeyne, 2013)
In this regard, it is tempting to speculate that the accumulation of succinate (the CII substrate in the TCA cycle) in GSH-deficient FoB (Fig
For bacterial GGTs, further examination of the X-ray crystal structure of EcGGT soaked in glutathione for varying time intervals also demonstrated the formation of -glutamyl enzyme intermediate, indicating that the second step is much slower than the first (Okada et al., 2006)
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